How polluted air can damage brain cells and cause memory loss
Air pollution will cause swelling and blockage of molecular routes, which leads to memory loss, finds studies.
According to a new study, air pollution has the ability to increase the risk of development of Alzheimer’s disease. With 99% of the world’s population breathing air, more than the World Health Organization (WHO) over the guidelines limit for air quality, this is the second major cause of annual deaths.
The study presented in the proceedings of the National Academy of Sciences highlighted the effects of air pollution on brain cells.
With the pollution of vehicles, stubble burning, air coolers, and construction work being the main trigger of air pollution, researchers at scrips research have found that it can also contribute to diverse brain diseases as Alzheimer’s and Automatic besides asthma and heart disease.
Increased air pollution, pesticides, smoke, wildfire, and processed meat can replace brain cells by disrupting nerve cell connections.
La Jola, a clinical neurologist from California said, “We have disclosed how pollutants can contribute to memory loss and neurodynative disease. It can eventually give rise to new drugs that can eventually block these effects to better treating Alzheimer’s disease,” La Jola, California’s a clinical sacrificial Said.
He coined the scientific term “SNO-Timan” two decades ago, which is formed in the brain when enlarged nitrous oxide and sulfur aligned due to an increase in air pollution.
This can lead to some forms of cancer, autism, Alzheimer’s disease, Parkinson’s disease and other conditions.
In this new study, the team focuses on the effects of brain blockage on the protein CRTC1, which helps regulate genes that are important to build and maintain between brain cells, an essential procedure for learning and long -term memory.
They found that the veins that are broken will be associated with other neurons, thus due to loss of memory. As we do age, inflammation increases with the level of nitrous oxide in the brain. This makes protein more susceptible to harmful brokerage of neurons.
He further created a genetically engineer version of CRTC1, which can no longer pass through breakdown. This restored the activation of the genes required for the connection between memory formation and neurons.
“We can almost completely completely include molecular routes in creating new memories,” Lipton said. “It suggests that it is a drugist target that can create a real difference in the treatment of Alzheimer’s and potentially other neurological diseases.”
The research team is now working on the development of drugs that can select some obstructing reactions including CRTC1.
